Vitamin B may be good for many things, but it won't slow mental decline as we age or prevent Alzheimer's disease
There may not only be a way to erase a memory, but also to restore it. Scientists have managed to erase a memory in rats before reactivating it, profoundly altering the animals' reactions to past events.
There may be a new technique to cure disease. Scientists have demonstrated a revolutionary new method in mice that could cure a wide range of human diseases--from cystic fibrosis to cataracts to Alzheimer's disease--that are caused by "misfolded" protein molecules.
There's a new drug out there that may help Alzheimer's patients in the near future. An experimental drug, called NitroMemantine, boosts brain synapses lost in Alzheimer's disease.
Researchers may have made a breakthrough when it comes to understanding the onset of Alzheimer's disease. They've discovered a catalytic trigger when the fundamental structure of a protein molecule changes to cause a chain reaction that leads to the death of neurons in the brain.
A new study suggests that Vitamin-B could help prevent Alzheimer's disease by reducing the loss of grey matter in vulnerable regions of the brain.
Victims of skin cancer may be less likely to develop Alzheimer's disease, according to a study published in the journal Neurology. Those with skin cancer were 80 percent more likely to develop Alzheimer's disease than those who did not have any skin cancer.
A drug developed by scientists at the Salk Institute for Biological Studies may be helpful reversing memory deficits and slowing Alzheimer's disease in aged mice following short-term treatment.
A drug that was used to slow Alzheimer's disease has failed to slow mental decline or preserve physical function.
A new potential benefit of a molecule in green tea was found by researchers at the University of Michigan: preventing the misfolding of specific proteins in the brain, leading to plaque, that are associated with Alzheimer's disease.
Scientists developed a drug against Alzheimer's that reduces the amount of the accompanying plaques in the brain by a third and more than doubles the number of new nerve cells in a particular region of the brain associated with memory. Professor David Allsop and Dr Mark Taylor at Lancaster Universit...